Coagulation Factor XIII-A A614T gene Variation is Suggestive of Founder Effect in Iranian Patients with Severe Congenital Factor XIII Deficiency

Authors

Majid Naderi1, Shadi Tabibian2, Shaban Alizade2, Zahra Sadat Abtahi3, Akbar Dorgalaleh4,*
1Department of Pediatrics Hematology & Oncology, Ali Ebn-e-Abitaleb Hospital Research Center for Children and Adolescents Health [RCCAH], ZahedanUniversity of Medical Sciences, Zahedan, Iran
2epartmentof Hematologyand Blood Transfusion, Allied Medical School, TehranUniversity of Medical Sciences, Tehran, Iran
3Departmentof Hematologyand Blood Transfusion, Allied Medical School, TehranUniversity of Medical Sciences, Tehran, Iran
4Department ofHematology and Blood Transfusion,School of Allied Medical Sciences,Iran University of Medical Sciences,Tehran, Iran.
*Corresponding Author: Department of Hematologyand Blood Transfusion, Allied Medical School, IranUniversity of Medical Sciences, Tehran, Iran Email: [email protected]

Journal of Cellular & Molecular Anesthesia:Vol. 1, issue 1; e150242
Published online:Dec 22, 2015
Article type:Original Articles
How to Cite:Naderi M, Tabibian S, Alizade S, Abtahi ZS, Dorgalaleh A. Coagulation Factor XIII-A A614T gene Variation is Suggestive of Founder Effect in Iranian Patients with Severe Congenital Factor XIII Deficiency. J Cell Mol Anesth. 2016;1(1):e150242. doi: https://doi.org/10.22037/jcma.v1i1.10638

Abstract

Background: Factor XIII (FXIII) is a heterotetramer consisting of two subunits, FXIII-A and FXIII-B. Several common gene variations were observed in the FXIII-A gene with an obvious ethnic difference. This study assessed the prevalence of A614T as a common FXIII-A gene variation among Iranian patients with FXIII deficiency (FXIIID). Materials and Methods: This study was conducted on eighty Iranian unrelated individuals with FXIIID. Genotype analysis for FXIII-A A614T gene variation was performed for all individuals. Results: Molecular analysis of these Iranian populations revealed that all studied patients were homozygous for the T allele at codon 204 of the FXIII-A1 subunit. Conclusion: Present of T allele at codon 204 of FXIII-A1 subunit among all study population can be suggestive of founder effect. ?

References

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    References are in the PDF file of the article.

Copyright

© 2016, Author(s). This open-access article is available under the Creative Commons Attribution 4.0 (CC BY 4.0) International License (https://creativecommons.org/licenses/by/4.0/), which allows for unrestricted use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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