In Vitro Challenge using Thymoquinone on Hepatocellular Carcinoma (HepG2) Cell Line

Authors

Sohair A. Hassan1,*, Wafaa A. Ahmed2, Fayek M. Galeb3, Maha A. El-Taweel4, Farid A. Abu-Bedair1
1Department of Medicinal Chemistry, National Research Centre, Dokki, Giza, Egypt
2Department of Cancer Biology, NCI, Cairo University, Cairo, Egypt
3Department of Clinical Pathology Research Institute of Ophthalmology, Cairo University, Cairo, Egypt
4Department of Clinical Pathology, NCI, Cairo University, Cairo, Egypt
*Corresponding Author: Department of Medicinal Chemistry, National Research Centre, Dokki, Giza, Egypt. Email:[email protected]

IJ Pharmaceutical Research:Vol. 7, issue 4; 283-290
Published online:Nov 20, 2010
Article type:Research Article
Received:Dec 01, 2007
Accepted:Aug 01, 2008
How to Cite:Hassan SA, Ahmed WA, Galeb FM, El-Taweel MA, Abu-Bedair FA. In Vitro Challenge using Thymoquinone on Hepatocellular Carcinoma (HepG2) Cell Line. Iran J Pharm Res. 2022;7(4):e128603. doi: https://doi.org/10.22037/ijpr.2010.777

Abstract

Black seed (Nigella sativa) is considered as a biological response modifier. Thymoquinone (TQ) is the bioactive and the most abundant constituent of the volatile oil of this seed which has been shown to possess anti-inflammatory, antioxidant and anti-neoplastic effects. In this study, the effect of TQ on HepG2 cell line was investigated in an attempt to identify its potential mechanism of action. Cell viability and proliferation were assessed in presence of different concentrations of TQ, which revealed a remarkable inhibition of HepG2 cells by TQ in a dose dependant manner. TQ ability to induced apoptosis was determined by Flowcytometry and colorimetric measurement of Caspases 3 and 9. The apoptotic effect of TQ was much more dramatic after 12 h treatment and the activity of Caspases 3 and 9 was increased. Also, Flowcytometric analysis of cell cycle revealed an early G1/S arrest of cells, which is characteristic of apoptosis. It could be concluded that Thymoquinone is a promising anti-cancer agent for hepatocellular carcinoma.

Copyright

© 2022, Author(s). This open-access article is available under the Creative Commons Attribution 4.0 (CC BY 4.0) International License (https://creativecommons.org/licenses/by/4.0/), which allows for unrestricted use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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