Personality disorders (PD) are associated with ways of thinking and feeling about oneself and others that significantly and adversely affect how an individual functions in many aspects of life. Since the fifth edition of the diagnostic and statistical manual of mental disorders (DSM-5) has shifted to a single axis system, the arbitrary boundaries between PD and other mental disorders have removed (
1). Considering that DSM refers to adolescence as the outset of PD, suggesting that symptoms can be traced back in early ages. Supporting this point, some researchers have shown that specific features of borderline personality disorder (BPD), including self-harm, impulsivity and affective instability being detected in childhood, are predictors of BPD in adulthood. As a matter of fact, DSMâs wording continues to allow for the diagnosis of PD in child and adolescent populations (
2).
Considering the age variable, PD categories may be used for those children and adolescents that their personality features appear to be pervasive, persistent and are not confined to a specific developmental stage or an episode of an axis I disorder. To diagnose PD in individuals under 18 years old, the features must have been presented for at least one year (
1). The main defining element is that the BPD symptomatology over 1-year or longer must be severe and persistent enough to disturb adolescentâs daily functions. Many of clinical researchers and personality theorists proposed that manifestation of personality disorders can be identified during adolescence and even earlier and this has been mentioned in diagnostic criteria of DSM (
2).
Investigating the borderline personality (BP) etiology, cognitive theories are based on schemata, which is a set of generalizations about oneself, others and the world (
3). When these schemata are formed, induce advances in processing subsequent information, and consequently enforce more cognitive distortions, such as alternating between extremes of idealization and devaluation and identity disturbance, which are features of BPD (
4).
Some theorists suggest that emotional dysregulation is the core feature of BPD (
5). The bio-social model (Linhan, 1993) is the most influential theory of these theories. More specifically, it is believed that BPD emerges from reciprocal effect of biological vulnerability (extremist emotion temperament) and invalidating environment (grading from average invalidation to extremely high). During development, if an emotive inborn response issued from a child is not replied adequately by the caregiver; therefore, the ability to present self-controlling or inhibit inappropriate emotional responses will never be learnt and this leads to fluctuations among emotional suppression and extreme emotion manifestations. When intense emotional reactions persistently occur during months and years, emotional dysregulations begin to become personality features resulting in social isolation and unstable relationships. Although these features are unadaptive as a result of emotion regulation and avoiding functionality, they are frequent and reinforcing. Consequently early vulnerabilities versus learning history, forms and preserves unadaptive aspects including cognitive, interpersonal, behavioral and emotional characteristics and eventually leads to BPD (
6).
Dissociation is disruption in the integrated functions of perception, consciousness, identity and memory. Patients with BPD undergo average to intense dissociative experiences, such as amnestic dissociation and depersonalization (
7). Researchers divided dissociation phenomena into the subtypes âpsychologicalâ (such as derealization and depersonalization) and âsomatoformâ (such as analgesia and tonic immobility). Stiglmayr et al. (
8) assert that patients with BPD experience somatoform on an average of 17% and psychological dissociation on an average of 20% in 24 hours. Generally, 33.3% of the patients with BPD experience severe somatoform and 41.7% experience psychological dissociation (
8).
Indeed, Barnow et al. (
9) acknowledge that dissociation canât be simply regarded as a learned strategy to reduce emotional involvement, but must be regarded as a dysfunctional strategy to regulate emotions, also inducing stress and autonomic output. This is supported by studies have demonstrated that dissociation leads to deficit in emotional regulation and emotion identification (
10), and that dissociative processes avoids emotional learning (
11). Moreover, Kleindienst et al. (
12) showed that dissociation predicts inefficient therapeutic consequences in patients with BPD under a dialectical behavioral therapy. In sum, it seems reasonable that dissociative patients react with dissociation as a response to negative emotions in psychotherapy and suppress their emotional responses that conclude in undesirable outcome.
Severe against stressor factors define patients with BPD and those with suicidal and self-destructive attempt (
8). Clinicians are well aware of the fear of abandonment in patients with BPD. This may be the most outstanding feature of the disorder because it informs clinician about the disorganized attachment styles, which patients with BPD deal with. When individuals need solidarity, abandonment is a new internalization of an intolerable strange self-image followed by self-destruction. Suicide reflects imaginary destruction of the strange individual internally. Committing suicide in BPD patients mostly aims to anticipate the probability of abandonment, attempting to preserve the relationship (
13). Genetic factors also play an important role in BPD suicidal attempts. Moreover, environment-inheritance interactions explain that environment alter the form of suicide genetic factors (
14). Therefore, repeated suicides in patients with BPD have high prevalence, with a lifelong risk of 10% (
15). In adolescents suicide rate is 16.9% and the suicidal attempt rate is 8.5% (
16). The prevalence of nonsuicidal self-injury is also high, 38 - 67% in clinical samples and 10 - 44% in nonclinical population (
17).